Neurobehavioral dysfunction in a mouse model of Down syndrome: upregulation of cystathionine β-synthase, H2S overproduction, altered protein persulfidation, synaptic dysfunction, endoplasmic reticulum stress, and autophagy

Author: T Panagaki, L Janickova, D Petrovic, K Zuhra, T Ditrói et al.

Year Published: 2024

Media Outlet: GeroScience

Open Access? Yes

Matrix: Tissue

Biological Themes: Neurological function

Product(s): Global Discovery Panel

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Neurobehavioral dysfunction in a mouse model of Down syndrome: upregulation of cystathionine β-synthase, H2S overproduction, altered protein persulfidation, synaptic dysfunction, endoplasmic reticulum stress, and autophagy

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